What are the effects of Class I antiarrhythmic? Select all thatapply
a. Decreased inotropic effect
b. Blocking stimulation of beta receptors in the heart
c. Increased myocardial contractility
d. An anticholinergic effect
(Visited 3 times, 1 visits today)
Related posts:
- Question: Nonspecific Beta Blockers (block Both Beta- 1 And Beta -2 Receptors) Have Been Used As First-step Antihypertensive Agents. Because These Agents Block Beta-2 As Well As Beta -1 Receptors, Bronchospasm May Occur. Newer Agents Are Beta-2 Specific And Are Not Contraindicated In Asthmatic Patients. Give Some Examples And Your Explanations For The Benefits …
- Question: Which Of The Following Sets Of Changes Are Present In A Preterm Infant Born At 32 Weeks Of Gestation With Surfactant Deficiency, As Compared To A Normal Infant? Alveolar Surface Tension Pulmonary Compliance А Decreased Decreased B с D Decreased Decreased Increased Increased No Change Decreased E F G Increased Increased No Change Increased No Change …
- Question: QUESTION 7 Which Of The Following Decreases When The Sympathetic System Is Activated? Heart Rate Bronchiole Diameter Pupil Diameter Blood Flow To GIT QUESTIONS Nifedipine, Diltiazem, And Verapamil Act By: Reducing The Release Of Norepinephrine Blocking Sodium Channels Blocking Muscarinic Receptors Blocking Calcium Channels QUESTIONS Click Save And Submit …
- Question: The Nurse Is Preparing To Administer The Anticholinergic Medication (cholinergic Blocking Drug] Benztropine (Cogentin) To A Patient Who Has Parkinson’s Disease. The Nurse Understands That This Drug Works By? ( See Chapter 21, 9th Ed., Cholinergic Blocking Drugs, Page 324] 1) Enhancing Ability Of Acetylcholine To Activate Cholinergic Receptors 2) Preventing …
- Question: Activation Of Beta, Receptors Cause: Select One Or More: O A. Promotes The Release Of Renin By The Kidneys B. Increase In Heart Rate C. Relaxes Uterine Smooth Muscle D. Increased Conduction Velocity Through The SA Nod E. Decrease In Heart Rate F. Dilation Of The Blood Vessels In The Kidneys G. Increased Force Of Myocardial Contraction Question 27. Answer …
- Question: Dopamine Is An Effective Treatment For Heart Failure Due To Its Mechanism Of Enhancing Contractility Of The Heart. This Benefit (of Contractility) Is Best Reflected In The Drugs Ability To Activate Which Receptor?[See Ch 18, 9th Ed., Adrenergic Drugs, Table 18.1 For Adrenergic Receptor Sites] 1) Beta 2) Dopamine 3) Alpha 4) Muscarinic
- Question: Minute Ventilation? What Happens If Increased Or Decreased? How Can We Fix It? Alveolar Ventilation What Happens If Increased Or Decreased? How Can We Fix It? Bohr Equation What Happens If Increased Or Decreased? How Can We Fix It? Total Arterial Oxygen Content What Happens If Increased Or Decreased? How Can We Fix It?
- Question: Identify Configuration And Characteristics Of Sympathetic And Parasympathetic Synapses Andreceptors. State Names And General Characteristics Of Drugs Affecting The Autonomic Nervous System. Identify Effects Produced By Stimulation Of Alpha And Beta-adrenergic Receptors. Identify Effects Of Antiadrenergic Drugs, Mechanisms Of Action And Effects On Body …
- Question: Which Is The MOA For Montelukast? O It Excites Leukotriene Receptors Which Improves Bronchoconstriction. O It Inhibits Leukotriene Receptors Which Causes Bronchodilation. O It Excites Leukotriene Receptors Which Causes Bronchodilation. O It Inhibits Leukotriene Receptors Which Exacerbates Bronchoconstriction.
- Question: Asthma Why Are Beta Agonists Used To Treat Asthma – How Do They Work? Why Must These Drugs Be Given Locally? Prototype Drug For Short-acting Beta Agonists? Prototype Drug For Long-acting Beta Agonists? Side Effects For Beta Agonists? How Are Corticosteroids Used To Treat Asthma? What Is The Prototype Drug For Corticosteroids Used To Treat Asthma? What …
